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10.3390/antiox11040756 33 KimJ

A reduction in antioxidant enzymatic activity in patients with hypertension has been reported

In AECs, TLR4/NF-B activation contributes to a pro-ferroptotic intracellular environment by upregulating genes such as NOX1 and NOX4 (promoting ROS production) and ACSL4 (increasing PUFA-containing phospholipid substrates), while suppressing ferroptosis-protective genes like GPX4 through redox imbalance and inflammatory stress (8688)

This makes it an exceptionally safe option for long-term, gentle detoxification, particularly for individuals with chronic illnesses whose systems are already fragile and nutrient-depleted