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We next identify and map sites of cholesterol high occupancy and residence time on active versus inactive GLP-1Rs using coarse-grained molecular dynamics (cgMD) simulations, followed by a screen of key residues selected from these sites and detailed analyses of the effects of mutating one of these residues, Val229, to alanine on GLP-1R interactions with cholesterol, plasma membrane behaviours, clustering, trafficking and signalling in pancreatic beta cells and primary islets, unveiling an improved insulin secretion profile for the V229A mutant receptor

In addition, the transcription factor KLF11 inhibits the proliferation of lung adenocarcinoma (LUAD) cells and promotes their chemosensitivity by participating in the GPX4-related ferroptosis pathway (37)

Schaum, N