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Based on previous studies [8, 19, 20] it is conceivable that the increases in lean mass we observed for GSH + CIT in the present study could have occurred due to increases in muscle protein synthesis, and this could be linked to NO-induced increases in cGMP [19]

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By disrupting this interaction, the peptide releases p53 to trigger apoptosis selectively in senescent cells while sparing healthy cells (Baar et al., 2017)

Spermidine synthase catalyzes a condensation reaction between dcAdoMet and putrescine, producing spermidine and 5-methylthioadenosine